Sharla's Log

**Youngblood** · 12-20-2008 05:45 AM

lol good idea sticking to the ATT forum. It can get ugly out there.

I too am thinking on a forum break. Hopefully I don't need to have myself banned again to make it happen...but I'm not above it or beyond another self-imposed intervention. Sorry...only read that far before responding.

Knee is feeling a bit better already, so ty for asking...and I'll let Salty and Scrap or someone else answer the other stuff I know nothing about.

**Sharla** · 12-20-2008 06:10 AM

Good to hear the knee is feeling better

I'm house sitting at the moment so it's an easier time for me to have a forum break than usual but still now while visiting my mum i manage to end up on the computer which i think is frankly a little sick!

As for answering stuff you know nothing about I'm not sure of what makes you think i know anything about it and i'm talking about it so why not you too?

If you have ideas, comments or just a guess it will all help because i'll have to double check the literature or try something to work out what i think about it and that will teach me more too.

I think it's easy to miss key things because you look at things from one angle so hearing other people's angles on things is always good.

**Youngblood** · 12-20-2008 06:42 AM

no srsly, too weakbrained atm 2 even try 2 figure anything chemical related, and I want to hide in here for a while, or take a break. Maybe I should make u some kinda bet to see who can stay away the longest. Oh and Sourpuss, if you read this, quit wimping out

k im out.

**Sharla** · 12-20-2008 07:13 AM

Right well offer stands if you want to chip into the conversation (which so far I'm having exclusively with myself

) later.

So I've read that endurance training increases capillarization and I just assumed I'd find sprints were better for increasing stroke volume. Alas I found the opposite:

Endurance rather than sprint running training increases left ventricular wall thickness in female athletes

JournalEuropean Journal of Applied Physiology

Tomas Venckunas1

, Rasa Raugaliene1, 3, Birute Mazutaitiene2 and Sonata Ramoskeviciute1
(1) Department of Applied Physiology and Sports Medicine, Lithuanian Academy of Physical Education, Sporto str. 6, 44221 Kaunas, Lithuania(2) Department of Languages, Lithuanian Academy of Physical Education, Sporto str. 6, 44221 Kaunas, Lithuania(3) Institute of Cardiology, Kaunas University of Medicine, Sukileliu ave. 17, Kaunas, Lithuania
Accepted: 28 September 2007 Published online: 17 October 2007
Abstract Competitive athletics is often associated with moderate left ventricular (LV) hypertrophy. The aim of this study was to shed more light on the extent and type of cardiac hypertrophic response to different athletic conditioning in females. Standard two-dimensional M-mode and Doppler echocardiography was performed at rest in Caucasian female sprinters (n = 10) and long-distance runners (n = 10) of similar age (range 16–34 years), training experience (5–18 years) and competitive level, and in age-matched healthy female sedentary controls (n = 10). No differences in echocardiographic parameters were detected between female sprinters and sedentary controls (p > 0.05). Interventricular septum and LV wall (p < 0.05) were thicker, and LV mass was greater (p < 0.01) in long-distance runners as compared with sprinters or sedentary controls. Absolute LV diameter was not increased in long-distance runners (p > 0.05), though relative LV diameter was higher in long-distance runners as compared to sprinters (p < 0.05). As compared with controls, relative wall thickness (the sum of LV wall thickness and interventricular septum thickness divided by LV diameter) was higher (p = 0.004) in long-distance runners. Neither systolic nor diastolic LV parameters were different among the groups (p > 0.05). In conclusion, sprint running training has not been found to induce alterations in cardiac morphology or function at rest in female athletes. Cardiac mass in female long-distance runners is higher mainly due to myocardial wall thickening, while integral myocardial function at rest is not affected as a consequence of either this hypertrophy or sprint training.

As expected, the heart rate and the double product were
lower in endurance runners, which was probably due to
altered cardiac autonomic regulation and larger stroke
volume.

So does this mean that capillarization and stroke volume are most significantly increased by endurance training then to increase VO2 max endurance training is the only way to go?

Unless there is another component of VO2 max I haven't taken into account people?